Genes | Free Full-Text | Homologous Recombination as a Fundamental Genome Surveillance Mechanism during DNA Replication
Kai Neelsen – Senior Medical Writer – nspm ltd | LinkedIn
Pathogens | Free Full-Text | Tissue Pathogens and Cancers: A Review of Commonly Seen Manifestations in Histo- and Cytopathology
MAD2L2 promotes replication fork protection and recovery in a shieldin-independent and REV3L-dependent manner | Nature Communications
Replication-Coupled DNA Repair - ScienceDirect
Frontiers | Replication Fork Reversal and Protection
Leveraging the replication stress response to optimize cancer therapy | Nature Reviews Cancer
DNA double-strand break repair-pathway choice in somatic mammalian cells | Nature Reviews Molecular Cell Biology
Frontiers | Single-Molecule Techniques to Study Chromatin
IJMS | Free Full-Text | DNA2 in Chromosome Stability and Cell Survival—Is It All about Replication Forks?
JoF | Free Full-Text | Post-Translational Modifications of PCNA: Guiding for the Best DNA Damage Tolerance Choice
Full article: Building up and breaking down: mechanisms controlling recombination during replication
Frontiers | The Replication Stress Response on a Narrow Path Between Genomic Instability and Inflammation
RIF1 promotes replication fork protection and efficient restart to maintain genome stability | Nature Communications
Two replication fork remodeling pathways generate nuclease substrates for distinct fork protection factors | Science Advances
Frontiers | UFL1, a UFMylation E3 ligase, plays a crucial role in multiple cellular stress responses
Replication stress and cancer | Nature Reviews Cancer
Sequential role of RAD51 paralog complexes in replication fork remodeling and restart | Nature Communications
Super-resolution visualization of distinct stalled and broken replication fork structures | PLOS Genetics
WRN helicase safeguards deprotected replication forks in BRCA2-mutated cancer cells | Nature Communications
Genes | Free Full-Text | Replication-Associated Recombinational Repair: Lessons from Budding Yeast
Ubiquitylation at Stressed Replication Forks: Mechanisms and Functions: Trends in Cell Biology
53BP1 Mediates ATR-Chk1 Signaling and Protects Replication Forks under Conditions of Replication Stress | Molecular and Cellular Biology
